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• Front cover
• Contents
• Preface
• Editors
• Contributors
• Chapter 1. Type, Topography, and Sequelae of Neuropathological Changes Shaping Clinical Phenotype of
• Chapter 2. Evidence for Oxidative Damage in the Autistic Brain
• Chapter 3. Oxidative Stress and Neurotrophin Signaling in Autism
• Chapter 4. Genetics of Autism
• Chapter 5. Phenotypic Expression of Autism, Maternal Depression, and the Monoamine Oxidase-A Gene
• Chapter 6. Paraoxonase 1 Status, Environmental Exposures, and Oxidative Stress in Autism Spectrum Di
• Chapter 7. The Redox/Methylation Hypothesis of Autism: A Molecular Mechanism for Heavy Metal-Induced
• Chapter 8. Autism and Oxidative Stress: Evidence from an Animal Model
• Chapter 9. Neurotoxic Brainstem Impairment as Proposed Threshold Event in Autistic Regression
• Chapter 10. Abnormalities in Membrane Lipids, Membrane-Associated Proteins, and Signal Transduction
• Chapter 11. Mitochondrial Component of Calcium Signaling Abnormality in Autism
• Chapter 12. Inflammation and Neuroimmunity in the Pathogenesis of Autism: Neural and Immune Network
• Chapter 13. Possible Impact of Innate Immunity on Autism
• Chapter 14. Autism, Gastrointestinal Disturbance, and Immune Dysfunction: What Is the Link?
• Chapter 15. Possible Mechanism Involving Intestinal Oxytocin, Oxidative Stress, and Signaling Pathwa
• Chapter 16. Cytokine Polymorphisms in Autism: Their Role in Immune Alterations
• Chapter 17. Autism, Teratogenic Alleles, HLA-DR4 and Immune Function
• Chapter 18. Autism: The Centrality of Active Pathophysiology and the Shift from Static to Chronic Dy
• Chapter 19. A Reevaluation of the State of Autism Treatment: The Need for a Biopsychosocial Perspect
• Index
• Back cover